β‘ Synaptic Transmission
Focuses on the vesicular release of neurotransmitters across the synaptic cleft during neuronal communication.
Examines vesicle docking, fusion, and recycling mechanisms that sustain rapid neural signaling.
Differentiates between glutamatergic (excitatory) and GABAergic (inhibitory) synaptic transmissions in neural networks.
Analyzes how voltage-gated and ligand-gated ion channels regulate depolarization and repolarization phases.
Explores the strengthening and weakening of synapses in response to experience β a foundation of learning.
Studies reuptake transporters that remove neurotransmitters from the synaptic cleft to regulate signaling duration.
Investigates gap junctions allowing direct cytoplasmic flow of ions between neurons, enabling instantaneous communication.
Examines classic synaptic junctions that rely on neurotransmitter diffusion for signal transmission.
Explores how multiple synaptic inputs combine to determine whether a neuron will fire an action potential.
Analyzes how drugs and neuromodulators alter synaptic activity, influencing cognition and behavior.